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Registro completo
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Biblioteca (s) : |
INIA Treinta y Tres. |
Fecha : |
19/11/2015 |
Actualizado : |
08/03/2018 |
Tipo de producción científica : |
Actividades de Difusión |
Autor : |
JORNADA ANUAL ARROZ-SOJA, 1983, CIAAB-EEE, TREINTA Y TRES, UY.; CHEBATAROFF, N.; DEAMBROSI, E.; BLANCO, P.H.; ÁVILA, S.; MÉNDEZ, R.; YANG, C.W.; WANG, W.J.; LAVECCHIA, A. |
Afiliación : |
NICOLÁS CHEBATAROFF; ENRIQUE GERMAN DEAMBROSI CHURRUT, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; PEDRO HORACIO BLANCO BARRAL, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; MARIFLOR STELLA ÁVILA SILVA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; RAMÓN FELIPE MÉNDEZ LARROSA, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; ANDRES PASCUAL LAVECCHIA GONZALEZ, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay. |
Título : |
Arroz-soja: Resultados de la experimentación regional en cultivos 1982-83. |
Fecha de publicación : |
1983 |
Fuente / Imprenta : |
Treinta y Tres (Uruguay): CIAAB; EEE, 1983 |
Páginas : |
104 p. |
Idioma : |
Español |
Notas : |
Actividad realizada en Agosto de 1983. |
Contenido : |
ARROZ
Mejoramiento
Épocas de siembra. Ensayos varietales. Ensayos regionales varietales. Respuesta varietal a nitrògeno. Convenio de Cooperación Técnico Agrícola con la República de China. Situación actual y perspectivas del mejoramiento genético de arroz en Uruguay.
Manejo
Ensayos regionales de fertilización. Manejo de nitrógeno. Discusión general de fertilización. Tesis de graduación. Ensayos especiales. Control de maleza.
Fitopatologìa
Camas de infección. Propagaciòn y daño provocado por enfermedades del tallo. Evaluación de fungicidas a campo. Evaluación de fungicidas en laboratorio.
SOJA
Ensayos varietales. Fertilización.
EVALUACIÒN DE DOS SISTEMAS INTENSIVOS DE PRODUCCIÓN EN SUELOS ARROCEROS.
DATOS CLIMÁTICOS |
Palabras claves : |
SISTEMA ARROZ-SOJA; SISTEMAS INTENSIVOS. |
Thesagro : |
ARROZ; SOJA. |
Asunto categoría : |
F01 Cultivo |
URL : |
http://www.ainfo.inia.uy/digital/bitstream/item/5254/1/Arroz-Soja1982-1983.pdf
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Marc : |
LEADER 01562nam a2200277 a 4500 001 1053942 005 2018-03-08 008 1983 bl uuuu u00u1 u #d 100 1 $aJORNADA ANUAL ARROZ-SOJA, 1983, CIAAB-EEE, TREINTA Y TRES, UY. 245 $aArroz-soja$bResultados de la experimentación regional en cultivos 1982-83.$h[electronic resource] 260 $aTreinta y Tres (Uruguay): CIAAB; EEE$c1983 300 $a104 p. 500 $aActividad realizada en Agosto de 1983. 520 $aARROZ Mejoramiento Épocas de siembra. Ensayos varietales. Ensayos regionales varietales. Respuesta varietal a nitrògeno. Convenio de Cooperación Técnico Agrícola con la República de China. Situación actual y perspectivas del mejoramiento genético de arroz en Uruguay. Manejo Ensayos regionales de fertilización. Manejo de nitrógeno. Discusión general de fertilización. Tesis de graduación. Ensayos especiales. Control de maleza. Fitopatologìa Camas de infección. Propagaciòn y daño provocado por enfermedades del tallo. Evaluación de fungicidas a campo. Evaluación de fungicidas en laboratorio. SOJA Ensayos varietales. Fertilización. EVALUACIÒN DE DOS SISTEMAS INTENSIVOS DE PRODUCCIÓN EN SUELOS ARROCEROS. DATOS CLIMÁTICOS 650 $aARROZ 650 $aSOJA 653 $aSISTEMA ARROZ-SOJA 653 $aSISTEMAS INTENSIVOS 700 1 $aCHEBATAROFF, N. 700 1 $aDEAMBROSI, E. 700 1 $aBLANCO, P.H. 700 1 $aÁVILA, S. 700 1 $aMÉNDEZ, R. 700 1 $aYANG, C.W. 700 1 $aWANG, W.J. 700 1 $aLAVECCHIA, A.
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Registro original : |
INIA Treinta y Tres (TT) |
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| Acceso al texto completo restringido a Biblioteca INIA Las Brujas. Por información adicional contacte bibliolb@inia.org.uy. |
Registro completo
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Biblioteca (s) : |
INIA Las Brujas. |
Fecha actual : |
30/06/2023 |
Actualizado : |
30/06/2023 |
Tipo de producción científica : |
Artículos en Revistas Indexadas Internacionales |
Circulación / Nivel : |
Internacional - -- |
Autor : |
GIANNITTI, F.; GARCÍA, J. P.; ADAMS, V.; ARMENDANO, J.; BEINGESSER, J.; ROOD, J.; UZAL, F. A. |
Afiliación : |
FEDERICO GIANNITTI, INIA (Instituto Nacional de Investigación Agropecuaria), Uruguay; JORGE P. GARCÍA, Universidad Nacional del Centro de la Provincia de Buenos Aires, Tandil, Buenos Aires, Argentina; VICKI ADAMS, Monash University, Parkville, Victoria, Australia; JOAQUÍN I. ARMENDANO, Universidad Nacional del Centro de la Provincia de Buenos Aires, Tandil, Buenos Aires, Argentina; JULIANN BEINGESSER, California Animal Health and Food Safety Laboratoy, University of California at Davis, San Bernardino, CA; JULIAN I. ROOD, Monash University, Parkville, Victoria, Australia; FRANCISCO A. UZAL, Monash University, Parkville, Victoria, Australia. |
Título : |
Experimental acute Clostridium perfringens type D enterotoxemia in sheep is not characterized by specific renal lesions. |
Complemento del título : |
Infectious Disease - Original Article. |
Fecha de publicación : |
2023 |
Fuente / Imprenta : |
Veterinary Pathology. 2023, vol.60(4):412-419. https://doi.org/10.1177/03009858231171669 |
ISSN : |
0300-9858 (print); 1544-2217 (online). |
DOI : |
10.1177/03009858231171669 |
Idioma : |
Inglés |
Notas : |
Article history: First published online May 12, 2023. -- Corresponding Author: Francisco A. Uzal, California Animal Health and Food Safety Laboratory, School of Veterinary Medicine, University of California at Davis, 105 W CVentral Ave, San Bernardino, CA 92408, USA. Email: fauzal@ucdavis.edu -- Funding: This work was supported by grant R01 AI056177 from the National Institute of Allergy and Infectious Diseases (NIAID). Research at Monash University was also supported by funding provided by the Australian Research Council to the Australian Research Council Centre of Excellence in Structural and Functional Microbial Genomics (grant no. CE0562063). -- |
Contenido : |
ABSTRACT.- Type D enterotoxemia, caused by Clostridium perfringens epsilon toxin (ETX), is one of the most economically important clostridial diseases of sheep. Acute type D enterotoxemia is characterized by well-documented lesions in the nervous, cardiocirculatory, and pulmonary systems. However, discrepancies and confusion exist as to whether renal lesions are part of the spectrum of lesions of this condition, which is controversial considering that for many decades it has been colloquially referred to as "pulpy kidney disease". Here, the authors assess renal changes in an experimental model of acute type D enterotoxemia in sheep and evaluate the possible role of ETX in their genesis. Four groups of 6 sheep each were intraduodenally inoculated with either a wild-type virulent C. perfringens type D strain, an etx knockout mutant unable to produce ETX, the etx mutant strain complemented with the wild-type etx gene that regains the ETX toxin production, or sterile culture medium (control group). All sheep were autopsied less than 24 hours after inoculation; none of them developed gross lesions in the kidneys. Ten predefined histologic renal changes were scored in each sheep. The proportion of sheep with microscopic changes and their severity scores did not differ significantly between groups. Mild intratubular medullary hemorrhage was observed in only 2 of the 12 sheep inoculated with the wild-type or etx-complemented bacterial strains, but not in the 12 sheep of the other 2 groups. The authors conclude that no specific gross or histologic renal lesions are observed in sheep with experimental acute type D enterotoxemia. © The Author(s) 2023 MenosABSTRACT.- Type D enterotoxemia, caused by Clostridium perfringens epsilon toxin (ETX), is one of the most economically important clostridial diseases of sheep. Acute type D enterotoxemia is characterized by well-documented lesions in the nervous, cardiocirculatory, and pulmonary systems. However, discrepancies and confusion exist as to whether renal lesions are part of the spectrum of lesions of this condition, which is controversial considering that for many decades it has been colloquially referred to as "pulpy kidney disease". Here, the authors assess renal changes in an experimental model of acute type D enterotoxemia in sheep and evaluate the possible role of ETX in their genesis. Four groups of 6 sheep each were intraduodenally inoculated with either a wild-type virulent C. perfringens type D strain, an etx knockout mutant unable to produce ETX, the etx mutant strain complemented with the wild-type etx gene that regains the ETX toxin production, or sterile culture medium (control group). All sheep were autopsied less than 24 hours after inoculation; none of them developed gross lesions in the kidneys. Ten predefined histologic renal changes were scored in each sheep. The proportion of sheep with microscopic changes and their severity scores did not differ significantly between groups. Mild intratubular medullary hemorrhage was observed in only 2 of the 12 sheep inoculated with the wild-type or etx-complemented bacterial strains, but not in the 12 sheep of the other 2 ... Presentar Todo |
Palabras claves : |
Clostridium perfringens type D; Enterotoxemia; ETX; Experimental infection; Kidneys; PLATAFORMA DE INVESTIGACIÓN EN SALUD ANIMAL - INIA; Renal pathology; Sheep. |
Asunto categoría : |
L10 Genética y mejoramiento animal |
Marc : |
LEADER 03352naa a2200325 a 4500 001 1064216 005 2023-06-30 008 2023 bl uuuu u00u1 u #d 022 $a0300-9858 (print); 1544-2217 (online). 024 7 $a10.1177/03009858231171669$2DOI 100 1 $aGIANNITTI, F. 245 $aExperimental acute Clostridium perfringens type D enterotoxemia in sheep is not characterized by specific renal lesions.$h[electronic resource] 260 $c2023 500 $aArticle history: First published online May 12, 2023. -- Corresponding Author: Francisco A. Uzal, California Animal Health and Food Safety Laboratory, School of Veterinary Medicine, University of California at Davis, 105 W CVentral Ave, San Bernardino, CA 92408, USA. Email: fauzal@ucdavis.edu -- Funding: This work was supported by grant R01 AI056177 from the National Institute of Allergy and Infectious Diseases (NIAID). Research at Monash University was also supported by funding provided by the Australian Research Council to the Australian Research Council Centre of Excellence in Structural and Functional Microbial Genomics (grant no. CE0562063). -- 520 $aABSTRACT.- Type D enterotoxemia, caused by Clostridium perfringens epsilon toxin (ETX), is one of the most economically important clostridial diseases of sheep. Acute type D enterotoxemia is characterized by well-documented lesions in the nervous, cardiocirculatory, and pulmonary systems. However, discrepancies and confusion exist as to whether renal lesions are part of the spectrum of lesions of this condition, which is controversial considering that for many decades it has been colloquially referred to as "pulpy kidney disease". Here, the authors assess renal changes in an experimental model of acute type D enterotoxemia in sheep and evaluate the possible role of ETX in their genesis. Four groups of 6 sheep each were intraduodenally inoculated with either a wild-type virulent C. perfringens type D strain, an etx knockout mutant unable to produce ETX, the etx mutant strain complemented with the wild-type etx gene that regains the ETX toxin production, or sterile culture medium (control group). All sheep were autopsied less than 24 hours after inoculation; none of them developed gross lesions in the kidneys. Ten predefined histologic renal changes were scored in each sheep. The proportion of sheep with microscopic changes and their severity scores did not differ significantly between groups. Mild intratubular medullary hemorrhage was observed in only 2 of the 12 sheep inoculated with the wild-type or etx-complemented bacterial strains, but not in the 12 sheep of the other 2 groups. The authors conclude that no specific gross or histologic renal lesions are observed in sheep with experimental acute type D enterotoxemia. © The Author(s) 2023 653 $aClostridium perfringens type D 653 $aEnterotoxemia 653 $aETX 653 $aExperimental infection 653 $aKidneys 653 $aPLATAFORMA DE INVESTIGACIÓN EN SALUD ANIMAL - INIA 653 $aRenal pathology 653 $aSheep 700 1 $aGARCÍA, J. P. 700 1 $aADAMS, V. 700 1 $aARMENDANO, J. 700 1 $aBEINGESSER, J. 700 1 $aROOD, J. 700 1 $aUZAL, F. A. 773 $tVeterinary Pathology. 2023, vol.60(4):412-419. https://doi.org/10.1177/03009858231171669
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